Rhabdomyolysis following severe hypokalemia caused by Conn’s syndrome
Journal Title: Archives of Medical Science - Year 2007, Vol 3, Issue 3
Abstract
Rhabdomyolysis denotes a clinical and laboratory syndrome that results from a rapid breakdown of skeletal muscle cells. This releases potentially toxic muscle cell components into the circulation which may cause life-threatening complications including myoglobinuric acute renal failure, cardiac arrest or disseminated intravascular coagulation. During rhabdomyolysis the injured muscle leaks potassium leading to hyperkalemia. In primary aldosteronism, mineralocorticoid activity leads to hypokalemia which can be severe enough to cause rhabdomyolysis. We report an unusual case of rhabdomyolysis with hypokalemia due to Conn’s syndrome.
Authors and Affiliations
Anna Gonerska-Szadkowska, Joanna Wittych-Długosz, Jacek Makarewicz, Mirosław Szmidt, Andrzej Lewiński
Keywords
Related Articles
- EP ID EP82671
- DOI -
- Views 252
- Downloads 0
Cardiovascular risk factors: lipids and lifestyle changes
Neopterin and circulating adhesion molecules as prognostic markers in childhood asthma
Introduction: The purpose of this study was to determine the serum levels of neopterin and circulating adhesion molecules sICAM-1, sE-selectin and sL-selectin in children with stable asthma in order to investigate thei...
The Abbott LCx-MTB assay is more sensitive than culture for detection of Mycobacterium tuberculosis in respiratory specimens
Background: In countries with a high incidence of tuberculosis quick, reliable and cost effective testing for this disease is mandatory. With the introduction of new methods based on detection of amplificated DNA fragmen...
Pulmonary abscesses – aetiology and treatment. Ten-year experience of the Department of General and Thoracic Surgery in Lodz, Poland
Introduction: Primary lung abscess is a disease developing due to aspiration or embolization of the infected material from the lung tissue or on the basis of other diseases of different aetiology (secondary abscess). In...
TGF-β regulates invasive behavior of human pancreatic cancer cells by controlling Smad expression
Introduction: To investigate the role of Smads in tumor cell activation, we examined changes in Smad expression as well as changes in proliferative and invasive behaviors in transforming growth factor-β (TGF-β) – stimula...